Search results for " Exercise-Induced"

showing 5 items of 5 documents

Airway Responsiveness to Histamine in Patients Refractory to Repeated Exercise

1988

To investigate the mechanisms contributing to refractoriness in exercise-induced asthma (EIA), airway responsiveness to histamine was studied in eight asthmatic patients. Patients were included in the study on the basis of their refractory response to multiple exercise challenges. Incremental challenges with inhaled histamine were performed at rest and 40 minutes after single and paired exercise tests. The geometric mean histamine concentration required to produce a 20 percent fall in FEV1 (PC20) for the challenge after paired exercise test (4.34 mg/ml) was significantly higher (p greater than 0.001) than those for the challenges after a single exercise (1.05 mg/ml) and for the challenge at…

AdultMalePulmonary and Respiratory MedicineTime FactorsRefractory periodPhysical ExertionCritical Care and Intensive Care MedicineBronchial Provocation Testschemistry.chemical_compoundRefractoryForced Expiratory VolumeHumansMedicineAsthmatic patientIn patientAsthmabusiness.industryRespiratory diseasemedicine.diseaseAsthmaAsthma Exercise-InducedchemistryAnesthesiaExercise TestFemaleCardiology and Cardiovascular MedicinebusinessAirway responsivenessHistamineHistamineChest
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Effects of exercise training and montelukast in children with mild asthma

2008

Data from the general population suggest that habitual exercise decreases bronchial responsiveness, but the possible role of exercise in asthmatics is undefined. The leukotriene receptor antagonist montelukast decreases bronchial responsiveness and exercise-induced symptoms in asthmatic children. This randomized study in children with mild asthma evaluated the combined effects of aerobic training for 12 wk and montelukast or placebo on bronchial responsiveness (BHR) to methacholine, exercise-induced bronchoconstriction (EIB), inflammatory markers in exhaled breath condensate (EBC), and asthma exacerbations.Fifty children (mean age +/- SD: 10.2 +/- 2.4 yr) with mild stable asthma were random…

CyclopropanesMaleQuinolineAcetatesSettore BIO/09 - Fisiologiaimmune system diseasesMedicineOrthopedics and Sports MedicineAnti-Asthmatic AgentsChildMethacholine ChlorideLeukotrieneeducation.field_of_studyrespiratory systemExercise TherapyAsthma Exercise-InducedBreath TestsItalyExhalationAnesthesiaQuinolinesFemalemedicine.drugHumanmedicine.medical_specialtyBreath TestBronchoconstrictionPopulationPhysical Therapy Sports Therapy and RehabilitationPhysical exerciseSulfidesSettore MED/10 - Malattie Dell'Apparato RespiratorioInternal medicineAerobic exerciseHumansAnti-Asthmatic AgenteducationMontelukastAsthmabusiness.industryLeukotriene receptorAcetateBronchospirometrymedicine.diseaseAsthmarespiratory tract diseasesPhysical FitnessPhysical FitneExercise TestMethacholinebusiness
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Increased airway inflammatory cells in endurance athletes: what do they mean?

2003

Inflammatory cells are increased in the airways of endurance athletes, but their role in causing exercise-induced respiratory symptoms and bronchoconstriction, or their possible long-term consequences, are uncertain.To put the results of athlete studies in perspective, by analysing the pathogenesis of airway cell changes and their impact on respiratory function.Athletes of different endurance sports at rest showed increased airway neutrophils. Elite swimmers and skiers also showed large increases in airway eosinophils and lymphocytes, possibly related to chronic, exercise-related exposure to irritants or cold and dry air, respectively. Post-exercise studies reported variable responses of ai…

NeutrophilsNeutrophilOsmolar ConcentrationRespiratory MucosaEosinophilLeukocyteSettore MED/10 - Malattie Dell'Apparato RespiratorioNitric OxideEosinophilsAsthma Exercise-InducedLeukocyte CountCell Adhesion MoleculeLeukocytesPhysical EnduranceHumansLymphocyteLymphocytesBronchial HyperreactivityCell Adhesion MoleculesHumanSports
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Left ventricular hypertrophy or storage disease? the incremental value of speckle tracking strain bull's-eye

2017

Left ventricular hypertrophy (LVH) develops in response to a variety of physical, genetic, and biochemical stimuli and represents the early stage of ventricular remodeling. In patients with LVH, subclinical left ventricular (LV) dysfunction despite normal ejection fraction (EF) may be present before the onset of symptoms, which portends a dismal prognosis. Strain measurement with two-dimensional speckle tracking echocardiography (STE) represents a highly reproducible and accurate alternative to LVEF determination. The present review focuses on current available evidence that supports the incremental value of STE in the diagnostic and prognostic workup of LVH. When assessing the components o…

Radiology Nuclear Medicine and ImagingSpeckle tracking echocardiographyDisease030204 cardiovascular system & hematologyLeft ventricular hypertrophytwo-dimensional strain0302 clinical medicineCardiomegaly Exercise-Induced030212 general & internal medicineanabolic steroidSubclinical infectionamyloidosiEvidence-Based MedicineEjection fractionHypertrophic cardiomyopathyleft ventricular hypertrophyEchocardiographyCardiologyElasticity Imaging TechniquesHypertrophy Left VentricularRadiologyCardiomyopathiesCardiology and Cardiovascular MedicineHumanendocrine systemmedicine.medical_specialtyarterial hypertensionReproducibility of ResultSensitivity and SpecificityDiagnosis Differential03 medical and health sciencesElasticity Imaging TechniqueInternal medicinemedicineathlete's heartHumanscardiovascular diseasesVentricular remodelingspeckle tracking echocardiographyCardiomyopathiebusiness.industryReproducibility of ResultsStroke Volumeaortic stenosiImage Enhancementmedicine.diseasehypertrophic cardiomyopathyDifferential diagnosisMetabolism Inborn ErrorbusinessMetabolism Inborn Errors
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Special Article - Exercise-induced right ventricular injury or arrhythmogenic cardiomyopathy (ACM): The bright side and the dark side of the moon.

2020

There is still debate on the range of normal physiologic changes of the right ventricle or ventricular (RV) function in athletes. Genetic links to arrhythmogenic cardiomyopathy (ACM) are well-established. There is no current consensus on the importance of extensive exercise and exercise-induced injury to the RV. During the intensive exercise of endurance sports, the cardiac structures adapt to athletic load over time. Some athletes develop RV cardiomyopathy possibly caused by genetic predisposition, whilst others develop arrhythmias from the RV. Endurance sports lead to increased volume and pressure load in both ventricles and increased myocardial mass. The extent of volume increase and cha…

medicine.medical_specialtySports medicineVentricular Dysfunction RightCardiomyopathy030204 cardiovascular system & hematologyRisk AssessmentBoth ventriclesSudden cardiac death03 medical and health sciences0302 clinical medicineInternal medicinemedicineGenetic predispositionHumansGenetic Predisposition to Disease030212 general & internal medicineCardiomegaly Exercise-InducedPathologicalExerciseArrhythmogenic Right Ventricular DysplasiabiologyVentricular Remodelingbusiness.industryAthletesMyocardiummedicine.diseasebiology.organism_classificationAdaptation PhysiologicalFibrosismedicine.anatomical_structureDeath Sudden CardiacVentricleAthletesHeart Disease Risk FactorsCardiologyPhysical EnduranceVentricular Function RightCardiology and Cardiovascular MedicinebusinessProgress in cardiovascular diseases
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